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Submitted: 07 Mar 2018
Accepted: 04 May 2018
ePublished: 18 May 2018
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J Renal Endocrinol. 2018;4(1): e14.
  Abstract View: 1414
  PDF Download: 749

Case Report

Bronchopulmonary carcinoid tumor presenting as dexamethasone suppressible Cushing’s syndrome; a case report

Rezvan Salehidoost 1, Sara Sadrzadeh 2, Mohammadreza Khosravifarsani 2*

1 Isfahan Endocrine and Metabolism Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.
2 Cancer Prevention Research Center, Isfahan University of Medical Sciences, Isfahan, Iran
*Corresponding Author: *Corresponding Author: Mohammadreza Khosravi, Email: , Email: Khosravifarsani@med.mui.ac.ir

Abstract

Cushing’s syndrome is an endocrine condition with complex diagnostic pathways. Cortisol suppression from high-dose dexamethasone suppression tests usually points to the pituitary as the cause. We present a patient with high-dose dexamethasone suppressible Cushing’s syndrome from a bronchopulmonary carcinoid tumor. A 30-year-old male presented with signs and symptoms of Cushing’s syndrome. Cortisol and ACTH levels were significantly elevated. High-dose dexamethasone test suppressed cortisol production. However, no pituitary source was found. In chest-computed tomography a well-differentiated mass was seen, octreotide scan localized somatostatin-positive tumor in the same place, which revealed a bronchopulmonary carcinoid tumor. Bronchopulmonary carcinoid tumor should be kept in the mind in the differential diagnosis of Cushing’s syndrome with suppression of the high-dose dexamethasone test if a pituitary source is not localized.

Citation: Salehidoost R, Sadrzadeh S, Khosravifarsani M. Bronchopulmonary carcinoid tumor presenting as dexamethasone suppressible Cushing’s syndrome; a case report. J Renal Endocrinol. 2018;4:e14.  
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